тяжелая черепно-мозговая травма, системная воспалительная реакция, иммуносупрессия, цитокины, апоптоз, Medicine

The article presents the review of pathophysiological mechanisms for the formation of the secondary immunological failure in severe craniocerebral injury: a stress-induced change in the functioning the neurohumoral axis, increase in the levels of catecholamines, cytokines, and activation of apoptosis of lymphoid cells.